The kidneys function as blood cleanser. But they also help regulate blood pressure. When the kidneys are damaged, your blood pressure may gradually turn. This is why patients with renal insufficiency may suffer from high blood pressure. How to lower blood pressure in patients with renal failure? Read on and find more information on this topic.
Kidney failure and high blood pressure
The kidneys function to filter blood around the body. It also helps to release some kind of substance called "renin" that can help shrink the arterial wall. Blood pressure refers to the force of blood against the artery wall. Thus, when the arterial wall is reduced, the blood pressure rose. That is why patients with renal insufficiency may suffer from high blood pressure.
What are the risks of hypertension in patients with renal failure?
- To worsen the condition of kidney and kidney functions decrease.
- Besides increasing the risk of developing heart disease.
- That can cause changes in mental status such as confusion
- What can cause bleeding in the brain
- Can lead to eclampsia, aneurysm, chest pain, etc
Thus, high blood pressure requires patients with renal impairment taking treatment as soon as possible.
How to lower blood pressure in patients with renal failure?
First, you have to realize that is kidney failure that causes high blood pressure. Thus, you need to take treatments that can help repair damaged kidney tissues and improve kidney function.
Based on this goal, Micro-Chinese Medicine Osmotherapy is suggested, because it can help repair damaged kidney tissues and lower blood pressure naturally.
Second, it is suggested also herbal tea. There are specific herbal tea has the natural ability to fight hypertension. It can help lower blood pressure naturally. If you want a list of suggested herbs, you can leave a message below or send us an e-mail kidney-support@hotmail.com
2014年12月18日星期四
Diabetic nephropathy
1. What disease is ill?
Diabetic nephropathy is a type of traditional diagnosis (this is a type of secondary nephropathy) as traditional explanation is correct, is the complication of diabetic microvascular mainly is diabetic glomerulosclerosis, is a type of pathological changes of glomerular dominated by vascular damage .According to the current most advanced and cutting-edge cytology is the disease of diabetic glomerular basement membrane.
2. How sick Diabetic nephropathy?
Because insufficient insulin or insulin opposes resistant, lead blood sugar up, blood sugar rises, it can lead to renal hemodynamic changes in kidney, high cholesterol, hypertension, and hyperglycemia appears, while producing large amounts of lipid peroxidation free radicals, and increased glycosylated hemoglobin glycation end products generated, these causes can injure mesothelial cells in glomerular endothelial cells are injured, may attract inflammatory cells infiltrate the blood while pound inflammatory mediators. These inflammatory mediators can infiltrate between mesangial area, leading to extend mesangial cells, epithelial cell fusion process produces dense foot drops, epithelial cells detached from the basement membrane thickening, stenosis cavities tube basement membrane, cease blood kidney failure and lack of oxygen causes the barrier function is impaired glomerular nuclear electronegative then can not prevent dripping blood protein, the protein appears +; patient's condition progressed, glomerular basement membrane collagen type 4 nucleic acids messenger increased sugar ago that thickening base film. by last article, the formation of diffuse lesions, mesangial nodular glomerulosclerosis occurs at this time may appear urea nitrogen and creatinine rise in clinician, the theory of this disease, we easily know that a Kurdish incompletely disease is that "damage glomerular basement membrane ".The symptoms are proteinuria and pathological symptoms of this series.
3. Why you sick this disease?
The cause of diabetic nephropathy especially are primary or secondary (eg genetic factors and environmental factors, lack of absolute and relative insulin, insulin resistance, hyperglycemia, metabolic syndrome) while in the pathogenesis of immune regulation associated with dysfunction, causes blood sugar rises in body intrinsic glomerular cells a number of pathological changes, it leads thickening of the glomerular basement membrane, tube cavities stenosis, vasoconstriction, microcirculation failure leave the blood and oxygen kidney failure, the capillaries laglomerular around wounded and tubular are also inflammatory infiltration, starts renal fibrosis, the onset of intrinsic renal cell phenotype, extracellular matrizes integrates many, supplanting healthy intrinsic renal cells, development of glomerular sclerosis, necrosis. We therefore call diabetic glomerular basement membrane disease.
For more information on diabetic nephropathy these things, you can fill out the form below to send the item, we will receive your message within 12 hours to tell you what you need.
Diabetic nephropathy is a type of traditional diagnosis (this is a type of secondary nephropathy) as traditional explanation is correct, is the complication of diabetic microvascular mainly is diabetic glomerulosclerosis, is a type of pathological changes of glomerular dominated by vascular damage .According to the current most advanced and cutting-edge cytology is the disease of diabetic glomerular basement membrane.
2. How sick Diabetic nephropathy?
Because insufficient insulin or insulin opposes resistant, lead blood sugar up, blood sugar rises, it can lead to renal hemodynamic changes in kidney, high cholesterol, hypertension, and hyperglycemia appears, while producing large amounts of lipid peroxidation free radicals, and increased glycosylated hemoglobin glycation end products generated, these causes can injure mesothelial cells in glomerular endothelial cells are injured, may attract inflammatory cells infiltrate the blood while pound inflammatory mediators. These inflammatory mediators can infiltrate between mesangial area, leading to extend mesangial cells, epithelial cell fusion process produces dense foot drops, epithelial cells detached from the basement membrane thickening, stenosis cavities tube basement membrane, cease blood kidney failure and lack of oxygen causes the barrier function is impaired glomerular nuclear electronegative then can not prevent dripping blood protein, the protein appears +; patient's condition progressed, glomerular basement membrane collagen type 4 nucleic acids messenger increased sugar ago that thickening base film. by last article, the formation of diffuse lesions, mesangial nodular glomerulosclerosis occurs at this time may appear urea nitrogen and creatinine rise in clinician, the theory of this disease, we easily know that a Kurdish incompletely disease is that "damage glomerular basement membrane ".The symptoms are proteinuria and pathological symptoms of this series.
3. Why you sick this disease?
The cause of diabetic nephropathy especially are primary or secondary (eg genetic factors and environmental factors, lack of absolute and relative insulin, insulin resistance, hyperglycemia, metabolic syndrome) while in the pathogenesis of immune regulation associated with dysfunction, causes blood sugar rises in body intrinsic glomerular cells a number of pathological changes, it leads thickening of the glomerular basement membrane, tube cavities stenosis, vasoconstriction, microcirculation failure leave the blood and oxygen kidney failure, the capillaries laglomerular around wounded and tubular are also inflammatory infiltration, starts renal fibrosis, the onset of intrinsic renal cell phenotype, extracellular matrizes integrates many, supplanting healthy intrinsic renal cells, development of glomerular sclerosis, necrosis. We therefore call diabetic glomerular basement membrane disease.
For more information on diabetic nephropathy these things, you can fill out the form below to send the item, we will receive your message within 12 hours to tell you what you need.
2014年12月17日星期三
Diabetic nephropathy
1. What disease is ill?
Diabetic nephropathy is a type of traditional diagnosis (this is a type of secondary nephropathy) as traditional explanation is correct, is the complication of diabetic microvascular mainly is diabetic glomerulosclerosis, is a type of pathological changes of glomerular dominated by vascular damage .According to the current most advanced and cutting-edge cytology is the disease of diabetic glomerular basement membrane.
2. How sick Diabetic nephropathy?
Because insufficient insulin or insulin opposes resistant, lead blood sugar up, blood sugar rises, it can lead to renal hemodynamic changes in kidney, high cholesterol, hypertension, and hyperglycemia appears, while producing large amounts of lipid peroxidation free radicals, and increased glycosylated hemoglobin glycation end products generated, these causes can injure mesothelial cells in glomerular endothelial cells are injured, may attract inflammatory cells infiltrate the blood while pound inflammatory mediators. These inflammatory mediators can infiltrate between mesangial area, leading to extend mesangial cells, epithelial cell fusion process produces dense foot drops, epithelial cells detached from the basement membrane thickening, stenosis cavities tube basement membrane, cease blood kidney failure and lack of oxygen causes the barrier function is impaired glomerular nuclear electronegative then can not prevent dripping blood protein, the protein appears +; patient's condition progressed, glomerular basement membrane collagen type 4 nucleic acids messenger increased sugar ago that thickening base film. by last article, the formation of diffuse lesions, mesangial nodular glomerulosclerosis occurs at this time may appear urea nitrogen and creatinine rise in clinician, the theory of this disease, we easily know that a Kurdish incompletely disease is that "damage glomerular basement membrane ".The symptoms are proteinuria and pathological symptoms of this series.
3. Why you sick this disease?
The cause of diabetic nephropathy especially are primary or secondary (eg genetic factors and environmental factors, lack of absolute and relative insulin, insulin resistance, hyperglycemia, metabolic syndrome) while in the pathogenesis of immune regulation associated with dysfunction, causes blood sugar rises in body intrinsic glomerular cells a number of pathological changes, it leads thickening of the glomerular basement membrane, tube cavities stenosis, vasoconstriction, microcirculation failure leave the blood and oxygen kidney failure, the capillaries laglomerular around wounded and tubular are also inflammatory infiltration, starts renal fibrosis, the onset of intrinsic renal cell phenotype, extracellular matrizes integrates many, supplanting healthy intrinsic renal cells, development of glomerular sclerosis, necrosis. We therefore call diabetic glomerular basement membrane disease.
For more information on diabetic nephropathy these things, you can fill out the form below to send the item, we will receive your message within 12 hours to tell you what you need.
Diabetic nephropathy is a type of traditional diagnosis (this is a type of secondary nephropathy) as traditional explanation is correct, is the complication of diabetic microvascular mainly is diabetic glomerulosclerosis, is a type of pathological changes of glomerular dominated by vascular damage .According to the current most advanced and cutting-edge cytology is the disease of diabetic glomerular basement membrane.
2. How sick Diabetic nephropathy?
Because insufficient insulin or insulin opposes resistant, lead blood sugar up, blood sugar rises, it can lead to renal hemodynamic changes in kidney, high cholesterol, hypertension, and hyperglycemia appears, while producing large amounts of lipid peroxidation free radicals, and increased glycosylated hemoglobin glycation end products generated, these causes can injure mesothelial cells in glomerular endothelial cells are injured, may attract inflammatory cells infiltrate the blood while pound inflammatory mediators. These inflammatory mediators can infiltrate between mesangial area, leading to extend mesangial cells, epithelial cell fusion process produces dense foot drops, epithelial cells detached from the basement membrane thickening, stenosis cavities tube basement membrane, cease blood kidney failure and lack of oxygen causes the barrier function is impaired glomerular nuclear electronegative then can not prevent dripping blood protein, the protein appears +; patient's condition progressed, glomerular basement membrane collagen type 4 nucleic acids messenger increased sugar ago that thickening base film. by last article, the formation of diffuse lesions, mesangial nodular glomerulosclerosis occurs at this time may appear urea nitrogen and creatinine rise in clinician, the theory of this disease, we easily know that a Kurdish incompletely disease is that "damage glomerular basement membrane ".The symptoms are proteinuria and pathological symptoms of this series.
3. Why you sick this disease?
The cause of diabetic nephropathy especially are primary or secondary (eg genetic factors and environmental factors, lack of absolute and relative insulin, insulin resistance, hyperglycemia, metabolic syndrome) while in the pathogenesis of immune regulation associated with dysfunction, causes blood sugar rises in body intrinsic glomerular cells a number of pathological changes, it leads thickening of the glomerular basement membrane, tube cavities stenosis, vasoconstriction, microcirculation failure leave the blood and oxygen kidney failure, the capillaries laglomerular around wounded and tubular are also inflammatory infiltration, starts renal fibrosis, the onset of intrinsic renal cell phenotype, extracellular matrizes integrates many, supplanting healthy intrinsic renal cells, development of glomerular sclerosis, necrosis. We therefore call diabetic glomerular basement membrane disease.
For more information on diabetic nephropathy these things, you can fill out the form below to send the item, we will receive your message within 12 hours to tell you what you need.
2013年6月11日星期二
Hypertension-One of the Common Symptoms of Polycystic Kidney Disease
According to clinical researches, there are more than 50 percent of patients of Polycystic Kidney Disease (PKD) have the symptom of hypertension when their renal function is still normal. When the renal function decreases, the morbidity of hypertension will be higher. Then how does PKD cause the hypertension?
With the continuous enlargement of the cysts, they will compress the normal renal tissues and renal blood vessels, which will cause the limited blood-supply or even the ischemia and anemia of renal blood vessels and the whole kidney. in this case, the receptor of the kidney will make reactions to the ischemia and anemia by secreting more rennin, which will lead to the increase of the density of angiotension and the contracting of blood vessels. Then the hypertension occurs. While, hypertension is not just the symptom of PKD, it also will aggravate the condition of this disease and accelerate the development of this disease.
Therefore, we must attach great importance to hypertension and adopt effective therapy to treat hypertension. To treat hypertension, we must proceed from the resource. That is to say we must aim at treating the cysts. On the basis of this theory, we take advantage of the thought of curing inner disease by outer therapy of our traditional Chinese medicine and use the effective Chinese medicine which is processed by advanced technique and especially for PKD. After been superfinely flattered, the medicine with extremely strong bioactivity and targeting positioning ability can conduct treatment on the certain cyst lesion. This type of therapy possesses two curative effects, the first one is: expand the vessels on the vascular wall surround the PKD, accelerate the blood circulation of cyst wall, promote the permeability of cyst wall, promote the inner press decrease of cyst wall vessels, reverse the differential pressure caused by intravascular pressure higher than intracapsular pressure, accelerate the vessels adsorption of the cyst liquid. The second one is: the active substance of Micro-Chinese Medicine can restrain the secretion of cyst wall epithelial cell; prevent the continue expanding of bursa wall vessels and cyst.
By this therapy, on one hand we can treat the symptom of hypertension. On the other hand, we can treat the disease of PKD fundamentally. If you have other questions about hypertension or Micro-Chinese Medicine, you can send email us: kidneyfailurecn@hotmail.com or contact us on-line.
2013年4月18日星期四
Hypertension—A Symptom of Polycystic Kidney Disease
Hypertension usually happens in 30s of the patients of Polycystic Kidney Disease when the disease is developing rapidly. This symptom reminds many patients with PKD genes to choose a treatment early.
Why hypertension can be sign and symptom of polycystic kidney disease in the early stage?
Polycystic Kidney Disease, similar with many other chronic kidney diseases, shows no symptoms obviously. In some cases, hypertension can be the first symptoms of Polycystic Kidney Disease.
If your family members, especially your parents or grandparents have the history of Polycystic Kidney Disease, you’d better to screen your kidneys when you monitor the high blood pressure (herbs help prevent high blood pressure related to polycystic kidney disease) in your 30s when PKD progress rapidly.
Does Polycystic Kidney Disease lead to the hypertension?
Yes. This is why hypertension is one of the symptoms of Polycystic Kidney Disease. When patient is in his 30s, renal cysts will develop fast. When renal cysts are big enough, they will suppress the kidney tissues. When kidney arteries get involved, the kidneys will be supplied with less blood and oxygen. In this condition, kidneys will release some hormones which can raise the blood pressure levels. These hormones include rein, angiotensin and aldosterone.
How to control the first symptoms of Polycystic Kidney Disease—hypertension?
Firstly, the blood pressure should be lower quickly. Hypertension itself not only can put the patient into emergency, but also can aggravate the kidney damages. Hypertension and kidney damages are a pair of malignant circle. Current anti-hypertensive pills, such as AECI and ARB, can be used to control the blood pressure to avoid the further worsening.
Secondly, kidney damages should be repaired. As mentioned above, when kidney can not get sufficient blood and oxygen, it will stimulate the EAAS to elevate blood pressure. The root cause is the renal cysts, thereby, shrinking the renal cysts should be conducted. As long as the renal cysts are controlled well, kidney damages will be alleviated and the symptoms of Polycystic Kidney Disease will disappear spontaneously.
Once your family members are diagnosed with Polycystic Kidney Disease, you need to test your kidneys condition regularly. If you fail to monitor your kidneys, the symptoms of Polycystic Kidney Disease should be a sign to remind you, including hypertension, urinary tract infection, flank pain, back pain, blood in urine, vomiting, poor sleep etc.
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